


#XPLAN B17 STARTUP GENERATOR#
IF RESTARTING IN FLIGHT : turn generator off IF RESTARTING IN FLIGHT : feather the propeller Set NAV1 frequency as needed - PDI should come to life.Set the LOCK MIXTURE AFT (auto system ON)ī - SETTING SYSTEMS TO “READY” B.1 - TURNING ELECTRICALS ON.Toggle Prop speed handles all the way up and down.Toggle all 4 waste-gates from on (pushed in) to off (pulled out).Toggle torque limiter all the way round from MAX (pushed forward) to none (pulled all the way aft).Toggle Manifold Pressure Selector (MPS) all the way round.Check payload and fuel (menu Aircraft/Payload & Fuel) - Wing tanks (1 & 3) must be 100% filled before using central tank (2) - Consider 800lb/100nm or 1350lb/hr.Check trims : ailerons, rudder, elevators at neutral position.Lock the tail wheel (only unlock when ready to taxi).Set brake primer (pull back 25% for grass, 50% for hard surfaces).Systems are not configured to work properly if the plane is loaded with engines running.Ĭhecklist done to B.3.3 MUST be executed in 3d view with the mouse, NOT with shortcuts ! A - PREFLIGHT A.1 - IMMOBILIZATION OF B17G This plane is designed to be started from a "Cold & Dark" configuration. EMERGENCY : RESTARTING AN ENGINE IN FLIGHT (B.2.1).

Together, these findings challenge the notion that mechanisms for OD plasticity contribute to the alterations in circuitry that restore acuity in amblyopia. Abolishing ngr1 expression in adult mice also promotes recovery of acuity. Reciprocally, deleting ngr1 in thalamus is insufficient to rectify eye dominance but yields improvement of acuity to normal. Deleting ngr1 in excitatory cortical neurons permits recovery of eye dominance but not acuity. Ngr1 mutant mice display greater excitatory synaptic input onto both inhibitory and excitatory neurons with restoration of normal vision. However, here we demonstrate that plasticity of eye dominance and acuity are independent and restricted by the nogo-66 receptor ( ngr1) in distinct neuronal populations. A predominant model is that “reactivating” ocular dominance (OD) plasticity after the critical period is required to improve acuity in amblyopic adults. Degrading vision by one eye during a developmental critical period yields enduring deficits in both eye dominance and visual acuity.
